SCN5A Variant K317M

Summary of observed carriers, functional annotations, and structural context for SCN5A K317M. Data combine curated literature, international cohorts, and gnomAD observations.

Estimated LQT3 penetrance

1%

0/11 effective observations

Estimated BrS1 penetrance

25%

2/11 effective observations

Total carriers

1

0 BrS1 · 0 LQT3 · 1 unaffected

K317M is present in 1 alleles in gnomAD. This residue resides in a Mild_Hotspot region for Brugada syndrome and a Non_Hotspot region for LQT3.

Variant features alone are equivalent to phenotyping 2 individuals for Brugada syndrome and 0 individuals for LQT3.

In silico predictors

Variant-level computational predictors.
PROVEAN PolyPhen-2 BLAST-PSSM REVEL Penetrance Density BrS (%) Penetrance Density LQT3 (%)
-0.57 0.741 0.18 0.597 34 0

PROVEAN scores below -2 suggest deleterious impact. REVEL scores above 0.5–0.75 are often interpreted as likely pathogenic. PolyPhen-2 scores above 0.85 are typically pathogenic. Penetrance density summarises neighbouring residue risk (Kroncke et al. 2019).

Reported carrier data

Observed carriers by publication or cohort.
Source Year Carriers Unaffected LQT3 BrS1 Other Other Disease
Literature, cohort, and gnomAD 1 1 0 0
Variant features alone 15 13 0 2

Totals may differ from individual publications due to duplicate patients removed during curation.

Nearby variants

Neighbouring residues within 15 Ångström provide structural context. Variants listed in the right-most column have been observed clinically or in gnomAD.

Previously observed variants near K317M.
Neighbour residue Distance (Å) Observed variants
343 12
277 13
342 11
346 11 E346K, E346G, E346X, E346D, E346D
319 7 G319S, G319R, G319C,
317 0 K317E, K317M, K317N, K317N,
282 14 R282C, R282H,
279 12
321 10 S321Y,
344 8 A344S,
340 14 R340W, R340Q,
345 8
322 14
280 15 C280Y,
318 6
281 11 V281M,
323 14
347 15
283 13
320 6 T320N,